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Studies in knock-out mice confirmed the biological action of [dA(2)]GLP-1/GcG which is related to its ability to target GIP, GLP-1, and glucagon receptors
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Engineering an improved IgG4 molecule with reduced disulfide bond heterogeneity and increased Fab domain thermal stability
GLP-1 medications work by reducing appetite and slowing digestion
doi: 10.1210/jcem.83.2.4551
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