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To test whether the effects of serotonin on glucose levels could be blocked by broad antagonism of serotonin receptor subtypes, the nonspecific antagonist cyproheptadine or an equal volume of saline was injected intraperitoneally 30 minutes before the mixed meal (Figure 6A)
It carries the fatty acids to the mitochondria of the cells to help produce energy
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Drug therapy through different pathologic mechanisms Alter metabolic substrates In 1962, Sodi-Pallares demonstrated in experimental models that glucose-insulin-potassium (GIK) therapy could enhance glycolytic flux and protect the myocardium from ischaemic damage [143]
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