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In isolated liver mitochondria, zinc reversibly inhibits KGDHC-dependent respiration, demonstrating a mechanistic link between matrix zinc levels and metabolic flux 86

In treatment methodology, restrictive intake of cysteine, glycine, histidine, methionine, tryptophan, and serine, amongst other amino acids, has shown positive outcomes in the pre-clinical phase and has potential for clinical implementation

In all the cases, the final concentration of seric zinc or/and selenium reached normal ranges after supplementation

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