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This perspective complements incretin-centered therapeutic frameworks by emphasizing metabolic flux proportionality as a determinant of long-term physiological resilience during pharmacological weight reduction

Sci Transl Med 5:209ra151

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It was reported that it could raise intracellular GSH by increasing SLC7A11 expression, efficiently leading to GPX4 enhancement and ferroptosis suppression in the ISO-induced injury model of H9C2 cells and rat myocardium [49]

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